International Review of Neurobiology (eBook)

Ronald J. Bradley, R. Adron Harris, Peter Jenner (Herausgeber)

eBook Download: PDF | EPUB

2004 | 1. Auflage
300 Seiten
Elsevier Science (Verlag)
978-0-08-049550-7 (ISBN)

International Review of Neurobiology is a well-respected series appealing to neuroscientists, clinicians, psychologists, physiologists, and pharmacologists. Led by an internationally renowned editorial board, this important serial publishes both eclectic volumes made up of timely reviews and thematic volumes that focus on recent progress in a specific area of neurobiology research. This volume is a cumulative subject index of volumes 26-50.

International Review of Neurobiology is a well-respected series appealing to neuroscientists, clinicians, psychologists, physiologists, and pharmacologists. Led by an internationally renowned editorial board, this important serial publishes both eclectic volumes made up of timely reviews and thematic volumes that focus on recent progress in a specific area of neurobiology research. This volume is a cumulative subject index of volumes 26-50.

Cover 1
Contents 6
Contents of Volumes 26–50 8
Subject Index 38
Contributor Index 332

Subject Index

Publisher Summary

This chapter lists the various terms discussed in Volume 58 of the series International Review of Neurobiology. It includes terms such as abnormal involuntary movement scale, accessory optic system, acetic anhydride, N-acetylaspartylglutamate, acetylcholine (ACh), acidic fibroblast growth factor, autoimmune mechanisms, and aversive memory.

A1 channel, 43:200

A77636, 48:111–112

AA, see Arachidonic acid

AAA, see Active analogue approach

ABC reaction, 47:230

Abdomen, musculature, 43:227–228

Abnormal Involuntary Movement Scale, tardive dyskinesia and, 31:300

Aβ plaques

in AD pathological diagnosis, 48:169–173

deposition, 48:182–184

Abrin, RNA, N-glycosidase toxin, 30:130–131

abrupt, see clueless

Absence epilepsy, 49:392–393

Absorption, zinc and, 31:152

ABT-431

and dyskinesias, 48:112

in PD tests, 48:109–110

aCC, see Anterior corner cell

Accessory optic system

binocular integration, 44:125–127

coordinate frame of reference, 44:129–136

cortical input, 44:45

decomposition, 44:124–125

description, 44:122–124

rotational stimulation, 44:127, 44:129

translational stimulation, 44:127, 44:129

ACE, see Angiotensin-converting enzyme

ACE inhibitors

clinical trials, 50:451–452

interventions

clinical, 50:419–421

experimental, 50:418

pharmacology, 50:423

and vascular basis for diabetic neuropathy, 50:416–418

Acetazolamide, anticonvulsant actions, astrocyte role, 30:182

Acetic anhydride, pretreatments, 47:114

N-Acetylaspartate

and creatine: ratio, 45:370

developmental patterns, 30:50–51

in diagnosis of Rasmussen’s syndrome, 45:187–188

phylogenetic and regional distribution in neural tissue, 30:46–49

N-Acetylaspartylglutamate

colocalization in different neuronal systems, 30:64(table)

degradation by N-acetylated, α-linked, acidic dipeptide-preferring peptidase, 30:75–81

developmental patterns, 30:50–51

electrophysiological effects, 30:81–82

in vitro, 30:84–87

in vivo, 30:82–85

endogenous, subcellular compartmentation, 30:75

enzymatic biosynthesis, 30:72–75

function, in vitro biochemical approaches, 30:87–88

as neuropeptide, 30:88

neurotransmission at glutamatergic pathways/synapses, 30:45–46, 30:89

pathways, lesion studies, 30:69–71

phylogenetic and regional distribution in neural tissue, 30:46–49

as precursor for transmitter glutamate, 30:90

receptors, identification, 30:91–92

ribosomal biosynthesis, 30:72–75

N-Acetylaspartylglutamate-like immunoreactivity

in brainstem and cerebellum (rodent), 30:58–65

colocalization in different neuronal systems, 30:64(table)

in forebrain nuclei and retina, 30:65–69

in spinal cord (rodent), 30:54–58

visualization, 30:51–52

visualization techniques, 30:52–54

N-Acetylated, α-linked, acidic dipeptide-preferring peptidase

N-acetylaspartylglutamate degradation, 30:75–81

amino acid and peptide inhibition, 30:77–79

regional distribution, 30:80(table)

structure-activity relationships, 30:78

Acetylation, neurotoxin-binding site and, 29:125

Acetylcholine (ACh), 31:82–84, 35:280–281, 35:376–377

alcohol and, 33:308, 33:333

application to CA1-cell apical dendrites, 30:265

ATPases, 35:305

P-type, 35:307–308

V-type, 35:305–307

clonidine and, 33:59, 33:96

antiwithdrawal effects, 33:75, 33:77

neurotransmitters, 33:68–72

pharmacologic actions, 33:87–90, 33:92

continuous leakage from motor nerve and muscle, 28:73–78

ACh receptors and, 28:77

botulinum toxin and, 28:76

curare-induced H-response and, 28:74–75

extracellular Ca2+ and, 28:74

mechanism of, 28:75–76

Na+, K+-ATPase and, 28:74–75

neurotrophic effect on muscle and, 28:77

physiological role, 28:76–78

control, 35:359

cell cultures, 35:360

genetic studies, 35:361

dopamine receptor subtypes and, 31:253

excitatory effects on neocortical neurons, 30:285–286

expression, 31:85–87, 31:90, 31:92–94

GABAA receptors and, 33:206

glial cells in activity-dependent plasticity and, 34:264

glutamate receptors and, 32:54, 32:55, 32:57, 32:100

invertebrate, 32:79, 32:95–98

mammalian, 32:61, 32:64

immunocytochemical location, 31:112, 31:113, 31:115, 31:122

inhibitory effects on neocortical neurons, 30:282–286

intermittent release from motor nerve

nonquantal, Ca2+-intensitive

4-aminoquinoline and, 28:71, 28:72

botulinum toxin and, 28:68, 28:70, 28:73

caffeine and, 28:70

cAMP and, 28:70, 28:79–80

giant mepp-inducing treatments and, 28:71

mechanism of, 28:72–73

physiological role, 28:73

slow spontaneous mepps, 28:68–72

temperature and, 28:69

quantal, Ca2+-sensitive, 28:59–68

botulinum toxin and, 28:67–68

cytoplasm-gate hypothesis, 28:63

drugs affecting Ca2+ entry and, 28:66–67

fast evoked mepps and, 28:60–62, 28:64, 28:66, 28:68, 28:70

physiological role, 28:67

temperature and, 28:69

vesicular hypothesis, 28:60–63

in IPN afferents, 28:164

nonquantal release

in frog, 28:278

mechanism of, 28:280–281

leech nervous system development and, 33:121, 33:162, 33:177

long-term potentiation and, 35:30

low-voltage fast activity, production in neocortex, 30:268

model

quantal release, 35:366–368

transmitter mobilization, 35:368–374

vesicular storage, 35:374–375

muscular dystrophy and, 29:8

myasthenic syndromes, 35:362–363

neocortical neuronal responses, 30:267

neuronal storage disease and, 29:228, 29:230

neuron development, 31:128, 31:129, 31:131, 31:132

neuropathy and, 31:399

neurotrophic factors and, 32:151, 32:156, 34:2, 34:6, 34:11, 34:17

olfaction and, 33:38

parkinsonism and, 31:5, 31:59

age of injury, 31:33

drugs, 31:41

future research, 31:48, 31:49, 31:51

lesions, 31:25, 31:26, 31:28

neuropathology,...

Erscheint lt. Verlag	26.1.2004
Sprache	englisch
Themenwelt	Medizin / Pharmazie ► Medizinische Fachgebiete ► Neurologie
	Naturwissenschaften ► Biologie ► Humanbiologie
	Naturwissenschaften ► Biologie ► Zellbiologie
	Naturwissenschaften ► Biologie ► Zoologie
ISBN-10	0-08-049550-8 / 0080495508
ISBN-13	978-0-08-049550-7 / 9780080495507

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