Autoimmunity versus Carcinogenesis in Tissue Injury

Preface; Predetermined Interplay of the Projected Malignant Transformation Step as Injury Non-Resolution; Systems of Active Immune Suppression as Integral Malignant Transformation in Infiltrating Glioma Cells; Systems of Failure of Immune Response to Early Carcinogenesis Recharacterize Such Early Carcinogenesis; Mirror Images of Geometric Pre-Setting of the Infiltrating Edges of High-Grade Gliomas; Failed Tumor Immuno-Antigenicity Is Expressed Suppression of an Autoimmunity in Defining Immune Privilege Status of the Lesion; Complex Effectiveness of General Immunity in Modulating the Specific Immune Response in Gliomas; Systems of Malignant Transformation as Integral Immune Nonresponse in Gliomagenesis; Immunodominance as Complex Patterned Interplay of Antigen Quantitation Phenomena in Pressure Immunity; Systems of Unstable Immunodominance of Antigen as Formulas for Central Immunosuppression in Tumor Cell Expansion; Failed Immunosurveillance as Failed Dendritic Cell Antigen PresentationSubstitution of Specific Tumor Antigenicity by Differentiation Associated Antigenicity; Projected Receptivity of Integral Gliomas to Malignant Transformation as Modeled by Dysfunctional Immunomodulation; Tumor Cell Identity as Projected by Immunotoxins within Heterogeneity and Hierarchical Systems of Progression; Dimensions of Reproducible Malignant Transformation as Brain Infiltration by Glioma; Infiltrativeness Attributes Project Schemes of Etiology and Pathogenesis in Sequential Malignant Transformations in Given Individual; Progressively Persistent Tumor Cell Growth and Recurrence and Spread as Interplay Dynamics between Individual Neoplastic Cells; Non-Response of the Immune System as Active Participant in Glioma Cell Genesis, Proliferation, and Spread in Normal Brain Tissues; Pathways of Non-Resolution in Targeting Tumor Cells by the Immune Response; Tumor Cell Antigenicity as Performance Hierarchy of Equilibrating Interactivity of Systems of Receptivity and Activation of Lymphocyte Subsets; Systems of Primary Regional Reformation in Tumor Origin and Mechanics: Regional versus Field Operability; Formulation of an Integral Antitumor Immune Response; Systems of Weak Immunogenicity as Originators of Early Spread and of Incremental Tumor Cell Growth and Proliferation; Index.