Multiple Sclerosis Immunology
Springer-Verlag New York Inc.
978-1-4939-4201-5 (ISBN)
Th17 cells in autoimmune inflammation and demyelination in the central nervous system.- Regulatory T cells in MS.- T-bet: A critical regulator of encephalitogenic T Cells.- Antigen-presenting cells in the Central Nervous System.- The role of B cells in multiple sclerosis.- Diet, gut flora and multiple sclerosis: current research and future perspectives.- GM-CSF in autoimmune inflammation of the central nervous system.- The role of Toll-like receptors in multiple sclerosis and experimental autoimmune encephalomyelitis.- Macrophages and microglia in experimental autoimmune encephalomyelitis and multiple sclerosis.- Genetics of multiple sclerosis.- Gene expression profiling and pathway analysis for identification of molecular targets in MS.- Systems biology for the study of multiple sclerosis.- Checkpoints in the development of pathogenic and regulatory T cells in Experimental Autoimmune Encephalomyelitis—a basis for current future interventions in MS.- Modeling MS in non-human primates.- A novel concept of treatment in MS: targeting both oligodendrocyte death and inflammatory processes by inhibiting poly (ADP-Ribose) polymerase.- Association of MS with other autoimmune diseases.- Current and future treatments of Multiple Sclerosis.- Targeting CD52 for the treatment of Multiple Sclerosis.- Haematopoietic stem cells for the treatment of MS.- Mesenchymal stem cells for the treatment of Multiple Sclerosis.
Erscheinungsdatum | 04.10.2016 |
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Zusatzinfo | 38 Illustrations, color; 8 Illustrations, black and white; XVII, 461 p. 46 illus., 38 illus. in color. |
Verlagsort | New York |
Sprache | englisch |
Maße | 155 x 235 mm |
Themenwelt | Studium ► 2. Studienabschnitt (Klinik) ► Humangenetik |
Studium ► Querschnittsbereiche ► Infektiologie / Immunologie | |
Schlagworte | autoimmune disease • Demyelination • immunotherapy • Multiple Sclerosis |
ISBN-10 | 1-4939-4201-8 / 1493942018 |
ISBN-13 | 978-1-4939-4201-5 / 9781493942015 |
Zustand | Neuware |
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